BOSCHELLE, Manuela

	Nome completo
	
					BOSCHELLE, Manuela
				
	Afferenza
	
					Dipartimento Universitario Clinico di Scienze mediche, chirurgiche e della salute
				
	SSD
	
					Settore MED/09 - Medicina Interna

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Risultati 1 - 3 di 3 (tempo di esecuzione: 0.001 secondi).

An animal model for the juvenile non-alcoholic fatty liver disease and non-alcoholic steatohepatitis

2016-01-01 Marin, Veronica; Rosso, NATALIA CAROLINA; DAL BEN, Matteo; Raseni, Alan; Boschelle, Manuela; Degrassi, Cristina; Nemeckova, Ivana; Nachtigal, Petr; Avellini, Claudio; Tiribelli, Claudio; Gazzin, Silvia

Can the interaction between the antimicrobial peptide LL-37 and alginate be exploited for the formulation of new biomaterials with antimicrobial properties?

2011-01-01 Toppazzini, Mila; Coslovi, Anna; Boschelle, Manuela; Marsich, Eleonora; Benincasa, Monica; Gennaro, Renato; Paoletti, Sergio

Fatty acids acutely enhance insulin-induced oxidative stress and cause insulin resistance by increasing mitochondrial reactive oxygen species (ROS) generation and nuclear factor-κB inhibitor (IκB)-nuclear factor-κB (NFκB) activation in rat muscle, in the absence of mitochondrial dysfunction.

2012-01-01 Barazzoni, Rocco; Zanetti, Michela; GORTAN CAPPELLARI, Gianluca; Semolic, ANNA MARIA; Boschelle, Manuela; Codarin, E.; Pirulli, Alessia; Cattin, Luigi; Guarnieri, Gianfranco

Titolo	Data di pubblicazione	Autori
An animal model for the juvenile non-alcoholic fatty liver disease and non-alcoholic steatohepatitis	1-gen-2016	MARIN, VERONICAROSSO, NATALIA CAROLINADAL BEN, MATTEORASENI, ALANBOSCHELLE, ManuelaDegrassi, CristinaNemeckova, IvanaNachtigal, PetrAvellini, ClaudioTIRIBELLI, CLAUDIOGAZZIN, SILVIA + -
Can the interaction between the antimicrobial peptide LL-37 and alginate be exploited for the formulation of new biomaterials with antimicrobial properties?	1-gen-2011	TOPPAZZINI, MILACOSLOVI, ANNABOSCHELLE, ManuelaMARSICH, ELEONORABENINCASA, MONICAGENNARO, RENATOPAOLETTI, SERGIO
Fatty acids acutely enhance insulin-induced oxidative stress and cause insulin resistance by increasing mitochondrial reactive oxygen species (ROS) generation and nuclear factor-κB inhibitor (IκB)-nuclear factor-κB (NFκB) activation in rat muscle, in the absence of mitochondrial dysfunction.	1-gen-2012	BARAZZONI, ROCCOZANETTI, MICHELAGORTAN CAPPELLARI, GIANLUCASEMOLIC, ANNA MARIABOSCHELLE, ManuelaCodarin, E.PIRULLI, ALESSIACATTIN, LUIGIGUARNIERI, GIANFRANCO + -

ArTS Archivio della ricerca di Trieste

BOSCHELLE, Manuela

An animal model for the juvenile non-alcoholic fatty liver disease and non-alcoholic steatohepatitis

Can the interaction between the antimicrobial peptide LL-37 and alginate be exploited for the formulation of new biomaterials with antimicrobial properties?

Fatty acids acutely enhance insulin-induced oxidative stress and cause insulin resistance by increasing mitochondrial reactive oxygen species (ROS) generation and nuclear factor-κB inhibitor (IκB)-nuclear factor-κB (NFκB) activation in rat muscle, in the absence of mitochondrial dysfunction.